Volume 5, Issue 3, June 2017, Page: 43-46
Gastric Outlet Obstruction Caused by a Triple Lesion: Eosinophilic Gastroenteritis, Brunner’s Gland Hyperplasia and Pancreatic Heterotopia in Unison
Apurva Sharma, Department of Medicine, Saint Peters University Hospital, New Brunswick, USA
Sonmoon Mohapatra, Department of Medicine, Saint Peters University Hospital, New Brunswick, USA
Kheng-Jim Lim, Department of Medicine, Division Gastroenterology and Hepatology, Rutgers Medical School Robert Wood Johnson, New Brunswick, USA
Abhijeet Chaubal, Department of Pathology, Saint Peters University Hospital, New Brunswick, USA
Arkady Broder, Department of Medicine, Division of Gastroenterology and Hepatology, Saint Peters University Hospital, New Brunswick, USA
Received: Mar. 1, 2017;       Accepted: Mar. 30, 2017;       Published: May 3, 2017
DOI: 10.11648/j.js.20170503.12      View  1963      Downloads  70
Abstract
Gastric outlet obstruction is a common entity described with numerous benign and malignant causes. Historically, benign causes include Peptic Ulcer disease (PUD); however recent cases have reported other causes including Eosinophilic Gastroenteritis (EGE), Brunner’s Gland Hyperplasia (BGH) and Pancreatic Heterotopia (PH). We present an unusual case of a 31 year old male patient with severe gastric outlet obstruction requiring surgical intervention caused by all three pathologies in unison – EGE, BGH and PH. The patient underwent surgical resection with distal gastrectomy and Billroth II reconstruction. Histology confirmed the three pathologies and post-operative recovery of the patient was uneventful.
Keywords
Eosinophilic Gastroenteritis, Brunner’s Gland Hyperplasia, Pancreatic Heterotopia
To cite this article
Apurva Sharma, Sonmoon Mohapatra, Kheng-Jim Lim, Abhijeet Chaubal, Arkady Broder, Gastric Outlet Obstruction Caused by a Triple Lesion: Eosinophilic Gastroenteritis, Brunner’s Gland Hyperplasia and Pancreatic Heterotopia in Unison, Journal of Surgery. Vol. 5, No. 3, 2017, pp. 43-46. doi: 10.11648/j.js.20170503.12
Copyright
Copyright © 2017 Authors retain the copyright of this article.
This article is an open access article distributed under the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/) which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
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